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During viral infections, intracellular GSH depletion occurs through multiple mechanisms and is necessary for viral replication

3920

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Following administration: Topical penetration through stratum corneum confirmed in skin permeability studies Systemic distribution following parenteral administration with preferential accumulation at injury sites Enhanced bioavailability compared to free copper due to peptide transport mechanisms Cellular uptake facilitated by specific copper transport pathways and receptors Studies using copper-64 radiolabeling showed preferential distribution to metabolically active tissues and sites of inflammation or injury, suggesting injury-targeted delivery mechanisms