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Rather than simply blocking pain signals, therapeutic peptides work at the cellular level to promote actual tissue healing and regeneration

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Similarly, in GPx1 −/− mice, deficiency of GPx1 increased ROS (as measured by DCF fluorescence of brain homogenates), lipid peroxidation, and protein carbonylation in brains exposed to the Aβ peptide, whereas forced overexpression of GPx1 in the GPx1 −/− mice attenuated these detrimental effects [124]