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in untreated moderate-to-severe MDD, cases can extend beyond a year without intervention

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This cardiomyopathy is characterized by dysfunction of cardiac mitochondria, a decrease in plasma selenium, decreased cardiac GPx1 expression, increased oxidative stress in the myocardium as reflected by an increase in 8-hydroxy-2-deoxy-guanosine content of nuclei, and decreased whole blood GPx1 activity [24, 25]