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These cardioprotective properties appear to be mediated at least partly through CD36 receptor binding, independent of the GHS-R1a/GH pathway, and represent one of the most promising avenues for future GHRP-6 research

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Additionally, the GH stimulatory effect on the liver to increase IGF-I secretion is accentuated by the direct GH effects in antagonizing insulin action and suppressing the anti-lipolytic actions of insulin (68)