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4 This should not be the case in the gastrointestinal tract where corticosteroids induce gastric lesions while prednisolone induced intestinal lesions (which were characteristic injuries) serve to establish prostaglandins cytoprotection in the intestine
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This should be a control of the release of glutamate in the synaptic cleft from presynaptic terminals to stimulate post-synaptic neurons (i.e., the NMDA receptors) and re-uptake from the cleft by the excitatory amino acid transporters (EAAT) into astrocytes, and glutamate/glutamine cycle within astrocytes [59]
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